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Turf Toe

Turf Toe
by RobertK83 » Fri Sep 30, 2016 4:10 pm

Diseases Gout - Turf Toe

Turf toe gets no respect. Let's take a closer look at the causes of turf toe and see how to treat it more effectively. Turf toe is actually a form of hallux limitus. Hallux limitus is classically described as pain and progressive decrease in the range of motion of the first metatarsal phalangeal joint (MPJ). The onset of hallux limitus is due to the following: :shock:

Functional Hallux Limitus

Biomechanical function that results in metatarsus primus elevatus and subsequent repetitive jamming of the first MPJ. Direct physical injury - injury to the articular cartilage or subchondral bone. These injuries may be due impaction injuries or hyperextension/flexion of the first MPJ.

The onset of turf toe always the same? Turf toe that is the result of functional hallux limitus is going to be insidious in onset. Functional hallux limitus will usually be seen in younger athletes as they attempt to increase their activity. It may not occur during the first athletic season, or the second, but when it does begin to cause pain, the onset will be more frequent and more severe, varying with activity. This profile of onset is simply due to the fact that the athlete is recreating the injury with every step. :lol:

Unger, K., Rahimi, F., Bareither, D., Muehleman, C. The Relationship Between Articular Cartilage Degeneration and Bone Changes of the First Metatarsophalangeal Joint. J. Foot Surg. 39:1 24-33, 2000

Ronconi, P., Monachino, P., Baleanu, P.M.,Favilli, G

Distal Oblique Osteotomy of the First Metatarsal for the Correction of Hallux Limitus and Rigidus Deformity. J. Foot Surg. 39:3, 154-160, 2000 This article will help you since it is a comprehensive study on Gout Joint :o.

Treatment of turf toe varies with the type of healthcare provider and includes the use of rest, shoe modifications, orthotics, steroid injections and surgery. The success of non-surgical care will vary with the degree of injury, the rate at which the injury is healing and how much osteoarthritis has occurred. We see varying degrees of success with orthotics that promote plantarflexion of the first ray, effectively treating metatarsus primus elevatus and peroneus longus dysfunction. Simple arch supports can make a significant difference in the symptoms of turf toe. Should our patient not respond to conservative care in a reasonable time period, we are not reluctant to suggest surgical revision to address the problem whether it be revisions of the joint defect, shortening of a long first metatarsal or structural revision of metatarsus primus elevatus. As mentioned before, the clinical appearance of dorsal lipping or visible radiographic changes are suggestive of moderately advanced osteoarthritis, a condition that is only repaired by joint revision or replacement. Isn't it wonderful that we can now access information about anything, including Gout Joint form the Internet without the hassle of going through books and magazines for matter!

Other conditions - synovitis, crystal deposition diseases such as gout, systemic arthritis, external physical influences such as Dupytren's contracture, etc.


When treating turf toe be sure to recognize the fact that there is no nerve innervation in articular cartilage. Pain associated with stage one turf toe (hallux limitus) is either synovial pain or bone pain. If we recognize that painful stage one turf toe (hallux limitus) may be due to bone pain, we then realize that turf toe should be treated aggressively to insure the long term viability of the joint. The more you read about Gout Joint, the more you get to understand the meaning of it. So if you read this article and other related articles, you are sure to get the required amount of matter for yourself.

What's the actual physical change that takes place in the joint with turf toe? As an easy analogy, consider the changes that takes place when an apple falls from a height and is damaged. The skin of the apple appears intact but the underlying pulp is damaged. In the case of turf toe (hallux limitus), think of the skin of the apple as the cartilage of the joint and the damaged pulp of the apple is the subchondral bone. Mild cases of turf toe (hallux limitus), result in little damage to the subchondral bone and will merely exhibit signs of inflammatory change within the joint. Most authors would refer to these cases as stage one turf toe (hallux limitus). More severe cases result in damage to the joint surface, the subchondral bone or both. These are the stage two and three cases of turf toe (hallux limitus) that show visible change on x-ray. As the subchondral bone becomes increasingly damaged, it will create an uneven supporting surface for the cartilage. An increase in activity results in uneven loading of the joint due to the compression injury of the subchondral bone. When doing an assignment on Gout Joint, it is always better to look up and use matter like the one given here. Your assignment turns out to be more interesting and colorful this way.

Think of turf toe (hallux limitus) as an isolated case of osteoarthritis limited to the first MPJ. Whether the injury is acute or due to repetitive loading, the result is a load that is applied to the subchondral bone that is greater than what the bone can tolerate. As the injury progresses, a series of micro fractures will develop in the subchondral bone. The typical soft spongy character of the metaphyseal bone changes to become brittle and hard. The result is that the articular cartilage looses its' underlying support and becomes susceptible to damage. Juxtachondral eburnation, osteophytes, lipping, spurring; call them what you like, but what you see on your x-ray is the slow progressive destruction of the joint.

Nomenclature: First MPJ - the big toe joint Metatarsus primus elevatus - a functional or structural position of the first metatarsal First metatarsal - the foot bone making up the proximal portion of the big toe joint Hallux - the great toe Learning about things is what we are living here for now. So try to get to know as much about everything, including Gout whenever possible.

Turf Toe Caused by a Direct Injury to the Joint May or May Not be Obvious

Athletes may not remember an incident of pain since they're often distracted by the event or game in which they're involved. The onset of direct injury to the joint may be abrupt, but also may be insidious becoming increasingly more painful as the season progresses. The joint pain will subside with rest only to recur with increased activity. It's not unusual to see symptoms of turf toe resolve in the off season only to recur with renewed exercise. People always think that they know everything about everything; however, it should be known that no one is perfect in everything. There is never a limit to learning; even learning about Diseases Gout.

References: Lombardi, C.M., Silhanek, A.D., Connolly, F.G., Dennis, L.N., Keslonsky, A.J. First Metatarsophalangeal Arthrodesis for Treatment of Hallux Rigidus: A Retrospective Study. J. Foot Surg. 40:3, 137-143, 2001 The best way of gaining knowledge about Gout Joint is by reading as much about it as possible. This can be best done through the Internet.

<hr> Symptoms: Turf toe that is the result of functional hallux limitus is going to be insidious in onset. Functional hallux limitus will usually be seen in younger athletes as they attempt to increase their activity. It may not occur during the first athletic season, or the second, but when it does begin to cause pain, the onset will be more frequent and more severe, varying with activity. This profile of onset is simply due to the fact that the athlete is recreating the injury with every step. Turf toe caused by a direct injury to the joint may or may not be obvious. Athletes may not remember an incident of pain since they're often distracted by the event or game in which they're involved. The onset of direct injury to the joint may be abrupt, but also may be insidious becoming increasingly more painful as the season progresses. The joint pain will subside with rest only to recur with increased activity. It's not unusual to see symptoms of turf toe resolve in the off season only to recur with renewed exercise. ;)

<hr> Anatomy: The great toe joint, or first metatarsal joint consists of two bones. The proximal bone is the first metatarsal and the distal is called the proximal phalanx or hallux. These two bone move against each other in an up and down motion through what we refer to as the sagital plane. Biomechanics: The motion of the great toe joint is dependant upon the plantarflexion of the first metatasal. If the first metatarsal is limitied in its' ability to plantarflex, the joint will jam resulting in funtional hallux limitus, which as we know is one of the causes of turf toe. Opportunity knocks once. So when we got the opportunity to write on Gout, we did not let the opportunity slip from our hands, and got down to writing on Gout.

Definition

Uric acid tests are tests that are done to measure the levels of uric acid in blood serum or in urine.

Purpose



You wish to learn more about Gout, Purines, or Uric Acid, check out Steve&#39;s s The Internet Guides. The Internet Guide: Uric Acid: http://gout.smartadssecrets.com/uric_acid :lol:

Steve Warshaw

Certified Personal Trainer and Nutritionist With over 15 years expereience developing training and nutrition programs for top level executives from companies such as Microsoft, Boeing, and Symetra Corp, Steve has established himself as a health and wellness expert. We were furnished with so many points to include while writing about Kidney Stone Formation that we were actually lost as to which to use and which to discard!

The Uric Acid Tests are Used to Evaluate the Blood Levels

Of uric acid for gout and to assess uric acid levels in the urine for kidney stone formation. The urine test is used most often to monitor patients already diagnosed with kidney stones, but it can also be used to detect disorders that affect the body&#39;s production of uric acid and to help measure the level of kidney functioning. The results of one reading this composition is a good understanding on the topic of Gout Uric Acid. So do go ahead and read this to learn more about Gout Uric Acid.


Abnormal results

The critical value for the blood test is a level of uric acid higher than 12 milligrams per deciliter (about 4 ounces).

Normal results

Blood test

Reference values for blood uric acid vary from laboratory to laboratory but are generally found within the following range: Male: 2.1-8.5 mg/dL; female: 2.0-6.6 mg/dL. Values may be slightly higher in the elderly. :roll:

Urine test

Reference values for 24-hour urinary uric acid vary from laboratory to laboratory but are generally found within the following range: 250-750 mg/24 hours.


Precautions

Blood test

Patients scheduled for a blood test for uric acid should be checked for the following medications: loop diuretics (Diamox, Bumex, Edecrin, or Lasix); ethambutol (Myambutol); vincristine (Oncovin); pyrazinamide (Tebrazid); thiazide diuretics (Naturetin, Hydrex, Diuril, Esidrix, HydroDiuril, Aquatensen, Renese, Diurese); aspirin (low doses); acetaminophen (Tylenol); ascorbic acid (vitamin C preparations); levodopa (Larodopa); or phenacetin. These drugs can affect test results. We have written a humorous anecdote on Uric Acid Levels to make it's reading more enjoyable and interesting to you. This way you learn there is a funny side to Uric Acid Levels too!

The Laboratory Should Also be Notified If the Patient Has

Had recent x-ray tests requiring contrast dyes. These chemicals increase uric acid levels in urine and decrease them in blood. Writing an article on Uric Acid Blood Levels was our foremost priority while thinking of a topic to write on. This is because Uric Acid Blood Levels are interesting parts of our lives, and are needed by us.

Key Terms

<dl> <dl> <dt>Fanconi&#39;s syndrome</dt> <dd>A rare disorder caused by vitamin D deficiency or exposure to heavy metals. </dd> <dt>Gout</dt> <dd>A metabolic disorder characterized by sudden recurring attacks of arthritis caused by deposits of crystals that build up in the joints due to abnormally high uric acid blood levels. In gout, uric acid may be overproduced, underexcreted, or both. </dd> <dt>Hyperuricemia</dt> <dd>Excessively high levels of uric acid in the blood, often producing gout. </dd> <dt>Purine</dt> <dd>A white crystalline substance that is one of the building blocks of DNA. Uric acid is produced when purine is broken down in the body. </dd> <dt>Uric acid</dt> <dd>A compound resulting from the body&#39;s breakdown of purine. It is normally present in human urine only in small amounts. </dd> <dt>Uricosuria</dt> <dd>Increased levels of uric acid in the urine. </dd> <dt>Wilson&#39;s disease</dt> <dd>A rare hereditary disease marked by the buildup of copper in the liver and brain, causing loss of kidney function.</dd> </dl> </dl>

Further Reading

For Your Information


Bio

Remember that it is very important to have a disciplined mode of writing when writing. This is because it is difficult to complete something started if there is no discipline in writing especially when writing on Uric Acid Blood Test.

You wish to learn more about Steve or to request training or nutrition information, please check out his website: http://www.bodybuiltbyvictoria.com.

Turf Toe Causes, Symptoms and Treatment | My Toe Hurts

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Certain Foods that are High in Purine May Increase the

Patient&#39;s levels of uric acid. These include kidneys, liver, sweetbreads, sardines, anchovies, and meat extracts. It is only through sheer determination that we were able to complete this composition on Uric Acid Levels. Determination, and regular time table for writing helps in writing essays, reports and articles. :lol:

Urine test

Patients should be checked for the following medications before the urine test: diuretics, aspirin, pyrazinamide (Tebrazid), phenylbutazone, probenecid (Benemid), and allopurinol (Lopurin). If the patient needs to continue taking these medications, the laboratory should be notified. It was at the spur of the moment that we ventured to write something about Uric Acid Blood Levels. Such is the amount of matter that is available on Uric Acid Blood Levels.

The Internet Guide: Gout - http://gout.smartadsscrets.com The Internet Guide: Purines - http://gout.smartadssecrets.com/purines.

Preparation

The uric acid test requires either a blood or urine sample. For the blood sample, the patient should be fasting (nothing to eat or drink) for at least eight hours before the test. The urine test for uric acid requires a 24-hour urine collection. The urine test does not require the patient to fast or cut down on fluids. Some laboratories encourage patients to drink plenty of fluids during the collection period. It is only because that we are rather fluent on the subject of Gout Uric Acid that we have ventured on writing something so influential on Gout Uric Acid like this! :D.

Increased production of uric acid may result from eating foods that are high in purine. Increased uric acid levels due to overproduction may also be caused by gout, by a genetic disorder of purine metabolism, or by metastatic cancer, destruction of red blood cells, leukemia, or cancer chemotherapy.

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Risks

Risks for the blood test are minimal, but may include slight bleeding from the puncture site, a small bruise or swelling in the area, or fainting or feeling lightheaded. :o.

Uric Acid is a Waste Product that Results from the

Breakdown of purine, a nucleic acid. (Nucleic acids are the building blocks of DNA.) Uric acid is made in the liver and excreted by the kidneys. If the liver produces too much uric acid or the kidneys excrete too little, the patient will have too much uric acid in the blood. This condition is called hyperuricemia. Supersaturated uric acid in the urine (uricosuria) can crystallize to form kidney stones that may block the tubes that lead from the kidneys to the bladder (the ureters).

Description

The uric acid blood test is performed on a sample of the patient&#39;s blood, withdrawn from a vein into a vacuum tube. The procedure, which is called a venipuncture, takes about five minutes. The urine test requires the patient to collect all urine voided over a 24-hour period, with the exception of the very first specimen. The patient keeps the specimen container on ice or in the refrigerator during the collection period.